Download Histological Typing of Salivary Gland Tumours by G. Seifert (auth.) PDF

By G. Seifert (auth.)

Knowledge of tumours of the salivary glands has complex think about­ ably within the 20 years that experience elapsed considering that paintings used to be all started at the first variation of Histological Typing of Salivary Gland Tumours. loads of info has been accumulated approximately newly de­ scribed tumour entities and the behaviour and diagnosis of the pre­ viously labeled tumours. Immunohistochemistry, cytophotometry, hybridization innovations, tissue tradition and chromosomal research have elevated our realizing of many tumours. Histological Typing Histological typing divides tumours of a given organ into differing kinds in response to their path of differentiation. even supposing this can often point out the underlying histogenesis of the tumour, it can be tough or most unlikely to spot the cellphone of foundation. word is taken of the constitution and serve as of mobilephone kinds, in addition to the final development trend of the tumour, with the purpose of matching those fea­ tures to these of a typical tissue present in a similar organ. the rules of the second one version of the WHO Histological Typing of Salivary Gland Tumours are in accordance with the subsequent axioms: - The class is oriented to the regimen paintings of the surgical pathologist. The inclusion of infrequent yet in actual fact outlined tumour en­ tities could be valuable to surgical pathologists consulting with clinicians.

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Sample text

Basal cell adenocarcinomas are low grade adenocarcinomas with a relatively good prognosis. Recurrence is relatively frequent, but metastasis is less common, usually to regional lymph nodes. Only one death has been documented in the literature at this time. This category of salivary gland tumour has only recently been clearly defined, and this entity was not included in the first edition of the classification. Although cumulative experience is small, one series has estimated its frequency at about 4% of all primary parotid gland carcinomas.

The development of a secondary carcinoma in a pre-existing pleomorphic adenoma can be observed in 3%-4% of all pleomorphic adenomas. The incidence of malignancy shows a correlation between the length of history of pleomorphic adenoma and the development of a carcinoma. 5% after more than 15 years. Concerningjnfiltrative growth and pathohistological differentiation, three subtypes can be distinguished: non-invasive carcinoma in pleomorphic adenoma, invasive carcinoma in pleomorphic adenoma and carcinosarcoma (true malignant mixed tumour).

Papillary . . . . . . Cystic lymphoid hyperplasia in AIDS 26 16 17 16 37 71, 72 29-31 31 29,30 122-124 Dysgenetic polycystic disease . . . 37 120 Lesion, benign lymphoepithelial -, tumour-like . . . . . . Lymphadenocarcinoma, sebaceous Lymphadenoma, sebaceous Lymphomas, malignant . . . 35 34 26 15 32 114,115 106-124 Malignant mixed tumour, see carcinoma, in pleomorphic adenoma Myoepithelioma -, malignant . 12 28 6, 7 80-83 Oncocytoma . Oncocytosis . 14 35 17-19 107-109 Papilloma, ductal -, intraductal -, inverted ductal 16 16 16 24-28 26 24,25 Salivary gland infarction, see sialometaplasia, necrotizing Sialadenitis, chronic sclerosing, of submandibular gland Sialadenoma papilliferum .

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