By J. Larry Jameson, Joseph Loscalzo
The authority of Harrison's in a convenient, full-color paperback committed completely to Nephrology and Acid-Base problems A Doody's middle name for 2011! four celebrity DOODY'S overview! "In the Harrison's culture, this booklet integrates pathophysiology with medical administration whereas outlining discoveries in genetics and molecular biology. It contains a few vital chapters that aren't present in comparable books: one on edition of the kidney to renal harm (including components and mechanisms of renal ailment development) and one on hyperuricemia and gout. The introductory bankruptcy at the simple biology of the kidney, masking renal improvement, functionality, and body structure, is especially good written. This publication meets the anticipated Harrison's criteria and analyzing it's a pleasant experience."--Doody's overview provider that includes the chapters on Nephrology and Acid-Base problems that seem within the landmark Harrison's ideas of inner drugs, 17e, this compact medical spouse provides all of the newest wisdom within the box, sponsored by means of the clinical rigor and reliability that experience outlined Harrison's. you can find insurance that displays the services of popular editors and participants -- provided in a carry-anywhere layout that makes it perfect for the study room, the wards, or the purpose of care. With its ease of use, quintessential diagnosis-speeding guidance, and acclaimed cutting-edge techniques, Harrison's Nephrology and Acid Base problems is a needs to for college students and clinicians alike. Harrison's Nephrology and Acid-Base issues promises: present, whole assurance of all significant renal-related shows and ailments, in addition to acid-base and electrolyte issues sixty three high-yield self-assessment Q&A for examination and board overview drawn from Harrison's rules of inner medication Self-Assessment and Board overview, 17e A well timed concentrate on the effect of clinical advances, with a glance at how genetics and molecular biology are reworking the sector New diagnostic and therapy ideas in keeping with proof from fresh scientific reviews Integration of pathophysiology with scientific administration issues in all the disease-oriented chapters worthy content material in print shape from the acclaimed Harrison's DVD worthy appendix that highlights laboratory values of medical value The Harrison’s area of expertise sequence is written through an identical world-renowned writer staff who introduced you Harrison’s rules of inner drugs, 17e: Anthony S. Fauci, MD; Eugene Braunwald, MD; Dennis L. Kasper, MD; Stephen L. Hauser, MD; Dan L. Longo, MD; J. Larry Jameson, MD, PhD; Joseph Loscalzo, MD, PhD
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Sample text
27 Hematuria, Pyuria, and Casts . . . . . . . . . . . . . 29 ■ Abnormalities of Urine Volume . . . . . . . . . . . . 30 Polyuria . . . . . . . . . . . . . . . . . . . . . 30 ■ Further Readings . . . . . . . . . . . . . . . . . 31 Normal kidney functions occur through numerous cellular processes to maintain body homeostasis. Disturbances in any of these functions can lead to a constellation of abnormalities that may be detrimental to survival.
A B FIGURE 4-12 Anti-GBM antibody-mediated glomerulonephritis. There is segmental necrosis with a break of the glomerular basement membrane and a cellular crescent (A), and A FIGURE 4-13 Amyloidosis. Amyloidosis shows amorphous, acellular expansion of the mesangium, with material often also infiltrating glomerular basement membranes, vessels, and in the interstitium, with apple-green birefringence by polarized immunofluorescence for IgG shows linear staining of the glomerular basement membrane with a small crescent at ∼1 o’clock (B).
0 and the urine is very concentrated or contaminated with blood. Tests to measure total urine concentration accurately rely on precipitation with sulfosalicylic or trichloracetic acids. Currently, ultrasensitive dipsticks are available to measure microalbuminuria (30–300 mg/d), an early marker of glomerular disease that has been shown to predict glomerular injury in early diabetic nephropathy (Fig. 3-3). The magnitude of proteinuria and the protein composition of the urine depend upon the mechanism of renal injury leading to protein losses.