By James G. Fox, Stephen Barthold, Muriel Davisson, Christian E. Newcomer, Fred W. Quimby, Abigail Smith
Ailments, the second one quantity within the 4 quantity set, The Mouse in Biomedical study, departs from the 1st version, via discussing particular disorder inflicting microorganisms, instead of the layout utilized in the 1st variation which mentioned infectious illnesses affecting particular organs and tissues. As such, the amount contains 26 chapters subdivided into RNA viruses and DNA viruses, in addition to bacterial, mycotic, and parasitic infections. those chapters not just offer updates on pathogenesis, epidemiology and prevention of formerly well-known murine pathogens, but additionally comprise info on newly well-known disease-causing organisms: mouse parvovirus, cilia linked breathing bacilli and Helicobacter spp. A separate class, which include three chapters, discusses zoonoses, tumor pathology of genetically engineered mice, and spontaneous ailments in widespread mouse lines.
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Extra resources for The Mouse in Biomedical Research, Volume 2, Second Edition: Diseases (American College of Laboratory Animal Medicine)
Example text
2002). The recognition of molecular latency in the absence of lowlevel chronic infection as the characteristic latent state in MCMV infection has allowed detailed studies of the mechanism of reactivation from latency and recurrence of infectious MCMV to be undertaken. As already discussed, it has been established that the recurrence of infectious MCMV following reactivation is controlled by the immune system, and CD8+ T cells play the leading role (Polic et al. 1998). However, the molecular reactivation of latent MCMV genomes is regulated by different means.
2001). Presumably, NK cells and T cells are protective through the reduction of the viral load that reaches the heart, while T cells also accumulate around the sites of myocardial infection and initiate disease. ROLE OF ANTIBODIES Antibodies may also play a role in myocarditis. In response to MCMV infection, both BALB/c and C57BL/6 mice produce antibodies that recognize myosin. However, only susceptible BALB/c mice produce antibodies during chronic myocarditis that interact with the cardiac isoform of myosin (O’Donoghue et al.
Cytomegalic cells are seen most commonly in the bile duct epithelium and infrequently in hepatocytes, indicating that the bile duct epithelium is an important site of HCMV replication in infants (Kosai et al. 1991). In immunocompetent adults, HCMV occasionally induces mononucleosis (Britt and Alford 1996), and in these cases the liver is often involved, with accompanying fever and moderately elevated transaminase activity. A mild, nonspecific hepatitis with a mononuclear cell infiltrate is observed (Ten Napel et al.